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Resources

Discover OWL Metabolomics contribution to scientific knowledge in our Resources section. We invite you to consult the publications in which we have participated, offering crucial insights into different therapeutic areas. Our library is here to help you with your research needs. We encourage you to explore our contributions with our extensive resources at your disposal. Use the filter options to search information by year of publication, therapeutic area, and/or disease.
Area
Aging Andrology Basic science Cardiology Cardiovascular Dermatology Ecology Endocrinology Fertility Gastroenterology Gynecology Hepatology Inmunology Metabolomics Neuroscience Nutrition Oncology Ophtalmology
Disease
Acute myeloid leukemia Aging Alcoholic Hepatitis Alzheimer’s Disease Amyotrophic lateral sclerosis Andrology Atopic dermatitis Bile acid diarrhoea Cardiovascular disease Cholangiocarcinoma Chronic venous disease Cirrhosis Colorectal cancer Cushing syndrome DILI Diabetes Diet in gestation Ecology Fertility Fuchs endothelial dystrophy Glaucoma HIV Hepatitis C Hepatoblastoma Hepatocellular carcinoma Hypercholesterolemia Inflammation Intervertebral disc degeneration Lipodistrophy Liver fibrosis Membrane biophysics Metabolomics Multiple sclerosis NAFLD NASH Nutrition Obesity Ovarian endometriosis Pancreatic cancer Pancreatic ductal adenocarcinoma Parkinson’s Disease Pharmacometabolomics Primary sclerosing cholangitis Prostate cancer Schwann cell myelination T2DM Toxicity Vaccinia Virus Infection Vascular disease Wilson’s disease
Year
2023 2022 2021 2020 2019 2018 2017 2016 2015 2014 2013 2012 2011 2010 2008
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Serum UPLC-MS/MS metabolic profiling in an experimental model for acute-liver injury reveals potential biomarkers for hepatotoxicity.
Metabolomics 2012;8(6):997-1011. / 2012
Area: Gastroenterology / Hepatology
Disease: DILI
Hepatic expression patterns of inflammatory and immune response genes associated with obesity and NASH in morbidly obese patients.
PLoS One 2010;5(10):e13577. / 2010
Area: Gastroenterology / Hepatology
Disease: NAFLD/NASH
Early effects of copper accumulation on methionine metabolism.
Cell Mol Life Sci. 2008;65(13):2080-90. / 2008
Area: Gastroenterology / Hepatology
Disease: Wilson’s disease
Loss of the glycine N-methyltransferase gene leads to steatosis and hepatocellular carcinoma in mice.
Hepatology 2008;47(4):1191-9. / 2008
Area: Oncology and Gastroenterology / Hepatology
Disease: NAFLD/NASH and Hepatocellular carcinoma
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