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Resources

Discover OWL Metabolomics contribution to scientific knowledge in our Resources section. We invite you to consult the publications in which we have participated, offering crucial insights into different therapeutic areas. Our library is here to help you with your research needs. We encourage you to explore our contributions with our extensive resources at your disposal. Use the filter options to search information by year of publication, therapeutic area, and/or disease.
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Aging Andrology Basic science Cardiology Cardiovascular Dermatology Ecology Endocrinology Fertility Gastroenterology Gynecology Hepatology Inmunology Metabolomics Neuroscience Nutrition Oncology Ophtalmology
Disease
Acute myeloid leukemia Aging Alcoholic Hepatitis Alzheimer’s Disease Amyotrophic lateral sclerosis Andrology Atopic dermatitis Bile acid diarrhoea Cardiovascular disease Cholangiocarcinoma Chronic venous disease Cirrhosis Colorectal cancer Cushing syndrome DILI Diabetes Diet in gestation Ecology Fertility Fuchs endothelial dystrophy Glaucoma HIV Hepatitis C Hepatoblastoma Hepatocellular carcinoma Hypercholesterolemia Inflammation Intervertebral disc degeneration Lipodistrophy Liver fibrosis Membrane biophysics Metabolomics Multiple sclerosis NAFLD NASH Nutrition Obesity Ovarian endometriosis Pancreatic cancer Pancreatic ductal adenocarcinoma Parkinson’s Disease Pharmacometabolomics Primary sclerosing cholangitis Prostate cancer Schwann cell myelination T2DM Toxicity Vaccinia Virus Infection Vascular disease Wilson’s disease
Year
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Dietary and genetic disruption of hepatic methionine metabolism induce acid sphingomyelinase to promote steatohepatitis.
Redox Biol. 2023;59:102596. / 2023
Area: Gastroenterology / Hepatology
Disease: NAFLD/NASH
The lipidomic and inflammatory profiles of visceral and subcutaneous adipose tissues are distinctly regulated by the SGLT2 inhibitor empagliflozin in Zucker diabetic fatty rats.
Biomed Pharmacother. 2023;161:114535. / 2023
Area: Gastroenterology / Hepatology & Endocrinology
Disease: NAFLD/NASH and T2DM
Serum lipidome unravels a diagnostic potential in bile acid diarrhoea.
Lewinska M, et al. Gut 2023;72(9):1698-708. / 2023
Area: Gastroenterology / Hepatology
Disease: Bile acid diarrhoea
Cholangiocarcinoma progression depends on the uptake and metabolization of extracellular lipids.
Hepatology 2022;76(6):1617-33. / 2022
Area: Oncology
Disease: Cholangiocarcinoma
Diagnostic accuracy of non-invasive tests for advanced fibrosis in patients with NAFLD: an individual patient data meta-analysis.
Gut 2002;71(5):1006-1019 / 2022
Area: Gastroenterology / Hepatology
Disease: NAFLD/NASH
One Carbon Metabolism and S-adenosylmethionine in Non-alcoholic Fatty Liver Disease Pathogenesis and Subtypes.
Livers 2022;2(4):243-57 / 2022
Area: Gastroenterology / Hepatology
Disease: NAFLD/NASH
Metabolic subtypes of nonalcoholic fatty liver disease patients exhibit distinctive cardiovascular risk profiles.
Hepatology 2022;76(4):1121-34. / 2022
Area: Gastroenterology / Hepatology
Disease: NAFLD/NASH
Inhibition of carnitine palmitoyl-transferase 1A in hepatic stellate cells protects against fibrosis.
J Hepatol. 2022;77(1):15-28. / 2022
Area: Gastroenterology / Hepatology
Disease: Liver fibrosis
A structurally engineered fatty acid, icosabutate, suppresses liver inflammation and fibrosis in NASH.
J Hepatol. 2022;76(4):800-11. / 2022
Area: Gastroenterology / Hepatology
Disease: NAFLD/NASH
The Treatment With the SGLT2 Inhibitor Empagliflozin Modifies the Hepatic Metabolome of Male Zucker Diabetic Fatty Rats Towards a Protective Profile.
Front Pharmacol. 2022;13:827033. / 2022
Area: Gastroenterology / Hepatology & Endocrinology
Disease: NAFLD/NASH and T2DM
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